Mechanism of the onset of pain syndrome

Pain may appear due to a crack in the disk
The development of the pathological process leads to the loss of intervertebral discs of elasticity and damping qualities. When the disease progresses, disks appear in the discs, the fibrous ring begins to bulge. Over time, hernias can form. Next to the intervertebral discs are nerve sprouts, blood vessels, spinal cord. Deformed vertebrae and hernia traumatize the nerve processes, which leads to their inflammation and provokes back pain, often radiating to other parts of the body.

The physiological mechanism of the pain syndrome in osteochondrosis is not fully understood. In a simplified form, it can be represented in the following form: the skin contains many nerve endings that react to any stimuli: injections, pressure, chemical factors, etc. Intensive exposure causes tissue damage, leading to irritation of the endings of nerve fibers – pain receptors. Signals from them are transmitted in the dorsal, and then in the brain, perceiving them as pain. Sensations in this case arise both in damage to nerve receptors and in damage to the nerves. The nature, localization and perception of pain vary depending on a number of factors. For example: degenerative changes lead to the infringement of the spinal nerve in the lumbar region, and the pain is felt in the foot – where the nerve begins. If the patient is concentrated on his feelings, besides is in a state of despair or depression, the pain can be subjectively perceived as more acute.

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